Big CWD press release coming

[Take Dead Aim]

Where are you getting this idea that only breeders are testing?

How many deer have you tested at your place?

[Take Dead Aim]

Where are you getting this idea that only breeders are testing?

Never said that. Please go reread. I know who and where testing takes places. I also know where it doesn't take place.

[Encinal]

How many deer have you tested at your place?

Probably 60

[Encinal]

Never said that. Please go reread. I know who and where testing takes places. I also know where it doesn't take place.

So you know it takes place in every RMU meeting criterion for a 95% confidence of detection at 5% prevalence in wild deer?

[hunt247]

Should just hire the green screen to fix the problem. Lot of experts here (not me). lol

[gtsticker]

Thanks for you advise. I'm more involved than most. I was the the Hueco Tank site when they killed the first CWD positive deer. I've been on site when two CWD positive native elk were killed in panhandle. I've helped with testing of over 100 mule deer. What else should I do to make you happy? I have first hand knowledge of the politics behind how this was handled. Like most govt agencies it was not dealt with in a smart manner. Yes that is just my opinion and probably not worth a penny to you. My opinion may turn out wrong and the state made all the right moves. Time will only tell. All that said I have been involved and hoping this passes without any major side effects to the deer population.

Not going to happen! Hoping and ignoring is what got Wyoming in trouble.
No one has all the answers, but like I said, every hunter should educate them selves so they do not become part of the problem.


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[Deerguy]

I think the only real question, that has nothing to do with hype and selling fear is, how many people have had a cross species issue from eating a CWD infected cervid.
The only thing that matter is the answer to that question.

And the answer is there are no known cases and no evidence to show there ever will be.

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That's not necessarily the ONLY question, but I agree it is the major question. If other wildlife species, and especially livestock, are impacted, that would certainly be bad and certainly have biological and economic implications. But, to your main point, we don't really know if people have been impacted yet. Prion-based diseases, or TSE’s, have an annoying habit of crossing species barriers and transforming into new diseases when some genetically-susceptible animals come in contact with prions. For example, CWD most likely started after deer came in contact with sheep scrapie prions. Scrapie has been around for a long, long, time and was thought to not be able to cross species barriers. But, when deer were placed in the pens, deer assimilated the prions and didn’t develop scrapie, but morphed into a new disease (CWD). CWD then became a disease that was easily transmissible to other cervids. Same thing happened with Mad Cow Disease, the bovine variety of TSE. Cows consumed prions and then morphed into MCD. And to carry it further, when bovid TSE (the prion vector for MCD) was introduced to people, a new disease was created, v-Crutchfield Jacobs Disease. CJD (TSE in people) has been known for a long time. But, the variant version is even different that the original after people consumed cows infected with MCD. So, its not as if there is no precedence for prion-based diseases to cross species barriers and create new diseases. Although nothing definitive has been shown yet, even the World Health Organization recommends not eating infected deer. The same thing happened with Mad Cow. There were people jumping up and down saying that people couldn’t get and it and all of the precautions were unnecessary. But then people got it. Plus, another major factor in determining if people are susceptible to CWD is the long incubation time for prion-based diseases (sometimes as long as 75% of the lifespan of the animal). So, its not as if you eat a CWD-infected animals and get sick the same day. It may take years or even decades to show causation. But, we do know that both humanized mice and the primate species most closely related to humans can be susceptible. So, since CWD is a relatively new disease, it is not surprising that no link, if one exists, has been determined as of yet.

[flounder9]

That's not necessarily the ONLY question, but I agree it is the major question. If other wildlife species, and especially livestock, are impacted, that would certainly be bad and certainly have biological and economic implications. But, to your main point, we don't really know if people have been impacted yet. Prion-based diseases, or TSE’s, have an annoying habit of crossing species barriers and transforming into new diseases when some genetically-susceptible animals come in contact with prions. For example, CWD most likely started after deer came in contact with sheep scrapie prions. Scrapie has been around for a long, long, time and was thought to not be able to cross species barriers. But, when deer were placed in the pens, deer assimilated the prions and didn’t develop scrapie, but morphed into a new disease (CWD). CWD then became a disease that was easily transmissible to other cervids. Same thing happened with Mad Cow Disease, the bovine variety of TSE. Cows consumed prions and then morphed into MCD. And to carry it further, when bovid TSE (the prion vector for MCD) was introduced to people, a new disease was created, v-Crutchfield Jacobs Disease. CJD (TSE in people) has been known for a long time. But, the variant version is even different that the original after people consumed cows infected with MCD. So, its not as if there is no precedence for prion-based diseases to cross species barriers and create new diseases. Although nothing definitive has been shown yet, even the World Health Organization recommends not eating infected deer. The same thing happened with Mad Cow. There were people jumping up and down saying that people couldn’t get and it and all of the precautions were unnecessary. But then people got it. Plus, another major factor in determining if people are susceptible to CWD is the long incubation time for prion-based diseases (sometimes as long as 75% of the lifespan of the animal). So, its not as if you eat a CWD-infected animals and get sick the same day. It may take years or even decades to show causation. But, we do know that both humanized mice and the primate species most closely related to humans can be susceptible. So, since CWD is a relatively new disease, it is not surprising that no link, if one exists, has been determined as of yet.

well said, i'm impressed deerguy!

also see;

*** These results would seem to suggest that CWD does indeed have zoonotic potential, at least as judged by the compatibility of CWD prions and their human PrPC target. Furthermore, extrapolation from this simple in vitro assay suggests that if zoonotic CWD occurred, it would most likely effect those of the PRNP codon 129-MM genotype and that the PrPres type would be similar to that found in the most common subtype of sCJD (MM1).***

http://www.tandfonline.com/doi/full/10.4161/pri.28124?src=recsys

http://www.tandfonline.com/doi/pdf/10.4161/pri.28124?needAccess=true

https://wwwnc.cdc.gov/eid/article/20/1/13-0858_article

8. Even though human TSE‐exposure risk through consumption of game from European cervids can be assumed to be minor, if at all existing, no final conclusion can be drawn due to the overall lack of scientific data. In particular the US data do not clearly exclude the possibility of human (sporadic or familial) TSE development due to consumption of venison. The Working Group thus recognizes a potential risk to consumers if a TSE would be present in European cervids. It might be prudent considering appropriate measures to reduce such a risk, e.g. excluding tissues such as CNS and lymphoid tissues from the human food chain, which would greatly reduce any potential risk for consumers. However, it is stressed that currently, no data regarding a risk of TSE infections from cervid products are available.

https://efsa.onlinelibrary.wiley.com/doi/full/10.2903/j.efsa.2018.5132

plus, there is a new TSE PRION DISEASE IN A NEW LIVESTOCK SPECIES, i.e. the camels that has spread since the initial announcement;

Prions cause fatal and transmissible neurodegenerative diseases, including Creutzfeldt-Jakob disease in humans, scrapie in small ruminants, and bovine spongiform encephalopathy (BSE). After the BSE epidemic, and the associated human infections, began in 1996 in the United Kingdom, general concerns have been raised about animal prions. We detected a prion disease in dromedary camels (Camelus dromedarius) in Algeria. Symptoms suggesting prion disease occurred in 3.1% of dromedaries brought for slaughter to the Ouargla abattoir in 2015–2016. We confirmed diagnosis by detecting pathognomonic neurodegeneration and disease-specific prion protein (PrPSc) in brain tissues from 3 symptomatic animals. Prion detection in lymphoid tissues is suggestive of the infectious nature of the disease. PrPSc biochemical characterization showed differences with BSE and scrapie. Our identification of this prion disease in a geographically widespread livestock species requires urgent enforcement of surveillance and assessment of the potential risks to human and animal health.

https://wwwnc.cdc.gov/eid/article/24/6/17-2007_article

https://camelusprp.blogspot.com/2020/11/tunisia-has-become-second-country-after.html

***> IMPORTS AND EXPORTS <***

***> SEE MASSIVE AMOUNTS OF BANNED ANIMAL PROTEIN AKA MAD COW FEED IN COMMERCE USA DECADES AFTER POST BAN

Saturday, April 14, 2018

Dromedary Camels Algeria Prion (Mad Camel Disease) TSE BSE MRR Import Export Risk Factors Excluding Grains and Plants

Dromedary Camels Algeria Prion (Mad Camel Disease) TSE BSE MRR Import Export Risk Factors Excluding Grains and Plants

(Grains and Plants Materials Could Harbor the Transmissible Spongiform Encephalopathy TSE Prion agent...TSS)

Dromedary Camels Algeria Prion (Mad Camel Disease) TSE BSE MRR Import Export Risk Factors Excluding Grains and Plants

http://camelusprp.blogspot.com/2018/04/dromedary-camels-algeria-prion-mad.html

https://camelusprp.blogspot.com/

also, PIGS, what about feral hogs, and how they tear up everything they come across, then think of feral hogs in a cwd endemic area, just thinking out of the box...

America BSE 589.2001 FEED REGULATIONS, BSE SURVEILLANCE, BSE TESTING, and CJD TSE Prion

so far, we have been lucky. to date, with the science at hand, no cwd transmitted to cattle, that has been documented, TO DATE, WITH THE SCIENCE AT HAND, it's not to say it has not already happened, just like with zoonosis of cwd i.e. molecular transmission studies have shown that cwd transmission to humans would look like sporadic cjd, NOT nvCJD or what they call now vCJD. the other thing is virulence and or horizontal transmission. this is very concerning with the recent fact of what seems to be a large outbreak of a new tse prion disease in camels in Africa. there is much concern now with hay, straw, grains, and such, with the cwd tse prion endemic countries USA, Canada. what is of greatest concern is the different strains of cwd, and the virulence there from? this thing (cwd) keeps mutating to different strains, and to different species, the bigger the chance of one of these strains that WILL TRANSMIT TO CATTLE OR HUMANS, and that it is documented (i believe both has already occured imo with scienct to date). with that said, a few things to ponder, and i am still very concerned with, the animal feed. we now know from transmission studies that cwd and scrapie will transmit to pigs by oral routes. the atypical bse strains will transmit by oral routes. i don't mean to keep kicking a mad cow, just look at the science;

***> cattle, pigs, sheep, cwd, tse, prion, oh my!

***> In contrast, cattle are highly susceptible to white-tailed deer CWD and mule deer CWD in experimental conditions but no natural CWD infections in cattle have been reported (Sigurdson, 2008; Hamir et al., 2006).

Sheep and cattle may be exposed to CWD via common grazing areas with affected deer but so far, appear to be poorly susceptible to mule deer CWD (Sigurdson, 2008). In contrast, cattle are highly susceptible to white-tailed deer CWD and mule deer CWD in experimental conditions but no natural CWD infections in cattle have been reported (Sigurdson, 2008; Hamir et al., 2006). It is not known how susceptible humans are to CWD but given that the prion can be present in muscle, it is likely that humans have been exposed to the agent via consumption of venison (Sigurdson, 2008). Initial experimental research suggests that human susceptibility to CWD is low and there may be a robust species barrier for CWD transmission to humans (Sigurdson, 2008), however the risk appetite for a public health threat may still find this level unacceptable.

https://assets.publishing.service.gov.uk/government/uploads/system/uploads/attachment_data/file/733407/DEFRA_QRA_TSE_in_cervids_June2018_v1.pdf

http://chronic-wasting-disease.blogspot.com/2012/08/susceptibility-of-cattle-to-agent-of.html

cwd scrapie pigs oral routes

***> However, at 51 months of incubation or greater, 5 animals were positive by one or more diagnostic methods. Furthermore, positive bioassay results were obtained from all inoculated groups (oral and intracranial; market weight and end of study) suggesting that swine are potential hosts for the agent of scrapie. <***

>*** Although the current U.S. feed ban is based on keeping tissues from TSE infected cattle from contaminating animal feed, swine rations in the U.S. could contain animal derived components including materials from scrapie infected sheep and goats. These results indicating the susceptibility of pigs to sheep scrapie, coupled with the limitations of the current feed ban, indicates that a revision of the feed ban may be necessary to protect swine production and potentially human health. <***

***> Results: PrPSc was not detected by EIA and IHC in any RPLNs. All tonsils and MLNs were negative by IHC, though the MLN from one pig in the oral <6 month group was positive by EIA. PrPSc was detected by QuIC in at least one of the lymphoid tissues examined in 5/6 pigs in the intracranial <6 months group, 6/7 intracranial >6 months group, 5/6 pigs in the oral <6 months group, and 4/6 oral >6 months group. Overall, the MLN was positive in 14/19 (74%) of samples examined, the RPLN in 8/18 (44%), and the tonsil in 10/25 (40%).

***> Conclusions: This study demonstrates that PrPSc accumulates in lymphoid tissues from pigs challenged intracranially or orally with the CWD agent, and can be detected as early as 4 months after challenge. CWD-infected pigs rarely develop clinical disease and if they do, they do so after a long incubation period. This raises the possibility that CWD-infected pigs could shed prions into their environment long before they develop clinical disease. Furthermore, lymphoid tissues from CWD-infected pigs could present a potential source of CWD infectivity in the animal and human food chains.

https://www.ars.usda.gov/research/publications/publication/?seqNo115=353091

https://www.ars.usda.gov/research/project/?accnNo=432011&fy=2017

https://www.ars.usda.gov/research/publications/publication/?seqNo115=337105

Friday, December 14, 2012

DEFRA U.K. What is the risk of Chronic Wasting Disease CWD being introduced into Great Britain? A Qualitative Risk Assessment October 2012

snip.....

In the USA, under the Food and Drug Administration's BSE Feed Regulation (21 CFR 589.2000) most material (exceptions include milk, tallow, and gelatin) from deer and elk is prohibited for use in feed for ruminant animals. With regards to feed for non-ruminant animals, under FDA law, CWD positive deer may not be used for any animal feed or feed ingredients. For elk and deer considered at high risk for CWD, the FDA recommends that these animals do not enter the animal feed system. However, this recommendation is guidance and not a requirement by law. Animals considered at high risk for CWD include:

1) animals from areas declared to be endemic for CWD and/or to be CWD eradication zones and

2) deer and elk that at some time during the 60-month period prior to slaughter were in a captive herd that contained a CWD-positive animal.

Therefore, in the USA, materials from cervids other than CWD positive animals may be used in animal feed and feed ingredients for non-ruminants.

The amount of animal PAP that is of deer and/or elk origin imported from the USA to GB can not be determined, however, as it is not specified in TRACES.

It may constitute a small percentage of the 8412 kilos of non-fish origin processed animal proteins that were imported from US into GB in 2011.

Overall, therefore, it is considered there is a __greater than negligible risk___ that (nonruminant) animal feed and pet food containing deer and/or elk protein is imported into GB.

There is uncertainty associated with this estimate given the lack of data on the amount of deer and/or elk protein possibly being imported in these products.

snip.....

36% in 2007 (Almberg et al., 2011). In such areas, population declines of deer of up to 30 to 50% have been observed (Almberg et al., 2011). In areas of Colorado, the prevalence can be as high as 30% (EFSA, 2011). The clinical signs of CWD in affected adults are weight loss and behavioural changes that can span weeks or months (Williams, 2005). In addition, signs might include excessive salivation, behavioural alterations including a fixed stare and changes in interaction with other animals in the herd, and an altered stance (Williams, 2005). These signs are indistinguishable from cervids experimentally infected with bovine spongiform encephalopathy (BSE). Given this, if CWD was to be introduced into countries with BSE such as GB, for example, infected deer populations would need to be tested to differentiate if they were infected with CWD or BSE to minimise the risk of BSE entering the human food-chain via affected venison. snip..... The rate of transmission of CWD has been reported to be as high as 30% and can approach 100% among captive animals in endemic areas (Safar et al., 2008).

snip.....

In summary, in endemic areas, there is a medium probability that the soil and surrounding environment is contaminated with CWD prions and in a bioavailable form. In rural areas where CWD has not been reported and deer are present, there is a greater than negligible risk the soil is contaminated with CWD prion. snip..... In summary, given the volume of tourists, hunters and servicemen moving between GB and North America, the probability of at least one person travelling to/from a CWD affected area and, in doing so, contaminating their clothing, footwear and/or equipment prior to arriving in GB is greater than negligible... For deer hunters, specifically, the risk is likely to be greater given the increased contact with deer and their environment. However, there is significant uncertainty associated with these estimates.

snip.....

Therefore, it is considered that farmed and park deer may have a higher probability of exposure to CWD transferred to the environment than wild deer given the restricted habitat range and higher frequency of contact with tourists and returning GB residents.

snip.....

https://web.archive.org/web/20170404125557/http://webarchive.nationalarchives.gov.uk/20130822084033/http://www.defra.gov.uk/animal-diseases/files/qra_chronic-wasting-disease-121029.pdf

***> READ THIS VERY, VERY, CAREFULLY, AUGUST 1997 MAD COW FEED BAN WAS A SHAM, AS I HAVE STATED SINCE 1997! 3 FAILSAFES THE FDA ET AL PREACHED AS IF IT WERE THE GOSPEL, IN TERMS OF MAD COW BSE DISEASE IN USA, AND WHY IT IS/WAS/NOT A PROBLEM FOR THE USA, and those are;

BSE TESTING (failed terribly and proven to be a sham)

BSE SURVEILLANCE (failed terribly and proven to be a sham)

BSE 589.2001 FEED REGULATIONS (another colossal failure, and proven to be a sham)

these are facts folks. trump et al just admitted it with the feed ban.

see;

FDA Reports on VFD Compliance

John Maday

August 30, 2019 09:46 AM VFD-Form 007 (640x427)

Before and after the current Veterinary Feed Directive rules took full effect in January, 2017, the FDA focused primarily on education and outreach. ( John Maday ) Before and after the current Veterinary Feed Directive (VFD) rules took full effect in January, 2017, the FDA focused primarily on education and outreach to help feed mills, veterinarians and producers understand and comply with the requirements. Since then, FDA has gradually increased the number of VFD inspections and initiated enforcement actions when necessary. On August 29, FDA released its first report on inspection and compliance activities. The report, titled “Summary Assessment of Veterinary Feed Directive Compliance Activities Conducted in Fiscal Years 2016 – 2018,” is available online.

https://www.fda.gov/media/130382/download

SUNDAY, SEPTEMBER 1, 2019

***> FDA Reports on VFD Compliance

https://bovineprp.blogspot.com/2019/09/fda-reports-on-vfd-compliance.html

[crc]

The risk isn’t worth it for designer deer

[flounder9]

Apparently there was positive CWD tests at a couple of huge breeding facilities which are connected to many more breeding facilities and release sites. Not good.

i finally got a recent copy of the CWD-trace facilities associated with the CWD-positive deer breeding facilities in Hunt and Uvalde Counties.

seems to date, there are 181 CWD-trace facilities associated with the CWD-positive deer breeding facilities in Hunt and Uvalde Counties, and some are out of state/country in Mexico (8).

i was told that in the coming weeks, some of the facilities will start testing for cwd, and those results will be forthcoming later on. i hope they don't flounder on depopulation efforts if any positives are found. sad for Mexico...

TUESDAY, APRIL 13, 2021

Implications of farmed-cervid movements on the transmission of chronic wasting disease

Conclusion

In conclusion, given that CWD transmission can occur through contact with infected body parts or through indirect contacts via contamination of feed and other fomites, understanding animal movements is critical for mitigating disease spread. Long distance commercial movements of cervids pose one risk for spread of CWD. This study approach can be used to understand disease transmission risks across the region and in North America in general.

TUESDAY, APRIL 13, 2021

Implications of farmed-cervid movements on the transmission of chronic wasting disease

https://www.sciencedirect.com/science/article/abs/pii/S0167587720301537?via%3Dihub

https://chronic-wasting-disease.blogspot.com/2021/04/implications-of-farmed-cervid-movements.html

WEDNESDAY, MARCH 31, 2021

Texas TPWD TAHC Chronic Wasting Disease Discovered at Deer Breeding Facilities in Hunt and Uvalde Counties

https://chronic-wasting-disease.blogspot.com/2021/03/texas-tpwd-tahc-chronic-wasting-disease.html

terry

[flounder9]

FRIDAY, MAY 14, 2021

Texas CWD TSE Prion Discovered at Deer Breeding Facilities in Matagorda and Mason Counties With 228 Positive To Date Total

“Regrettably, the gravity of this situation continues to mount with these new CWD positive discoveries, as well as with the full understanding of just how many other facilities and release sites across Texas were connected to the CWD positive sites in Uvalde and Hunt Counties,” said Carter Smith, Executive Director of TPWD.

https://chronic-wasting-disease.blogspot.com/2021/05/texas-cwd-tse-prion-discovered-at-deer.html

terry

[gtsticker]

How many is a ton? What percentage of the population killed is a ton? Sounds like some solid science there.

They’re testing 100% of dead deer from breeding facilities and the percentage of infected is infinitesimally small.

Kyle your an uniformed idiot that shoots your mouth off any time CWD is discussed. Go educate your dumb arse so you can have an intelligent conversation.


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[kyle1974]

Big CWD press release coming

Kyle your an uniformed idiot that shoots your mouth off any time CWD is discussed. Go educate your dumb arse so you can have an intelligent conversation.


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You should probably learn the difference between your and You’re when you get ready to criticize Intelligence. [emoji106]

As for what my post said, it’s true. Sorry if that hurt your feelings.

[justletmein]

FRIDAY, MAY 14, 2021

Texas CWD TSE Prion Discovered at Deer Breeding Facilities in Matagorda and Mason Counties With 228 Positive To Date Total

“Regrettably, the gravity of this situation continues to mount with these new CWD positive discoveries, as well as with the full understanding of just how many other facilities and release sites across Texas were connected to the CWD positive sites in Uvalde and Hunt Counties,” said Carter Smith, Executive Director of TPWD.

https://chronic-wasting-disease.blogspot.com/2021/05/texas-cwd-tse-prion-discovered-at-deer.html

terry

The Hunt facility underwent further DNA testing to confirm animal identification and origin, and on May 12 the DNA test results confirmed the deer’s connection to the premises.

Seems like a curious statement to add. I wonder if they tried to dodge the testing saying they didn't receive any deer from the Uvalde facility. Maybe they just had multiple sources and had to confirm which of the deer came from where?

[sharpstick35]

I would almost put money on the animal rights groups, (especially under the current administration), will use this to try and shut down hunting all together.