Originally posted by flounder9
View Post
-
LMAO! You're wanting to spread awareness of CWD scientific evidence, but if we don't understand it just move on. I'm not a CWD denier, and appreciate you sharing the information, but come on man. -
Agreed, I like science but it doesn't mean I speak it well.Originally posted by crc View PostComment
-
Keith Warren is nothing but a shill for whoever is throwing money his way.Originally posted by sectxag06 View PostComment
-
-
FRIDAY, JANUARY 04, 2019
TEXAS TPWD CONFIRMED CWD TSE PRION 3 WTD in Medina, Dallam, and Hartley Counties, and in 3 MD in Hudspeth, Hartley, and El Paso Counties
Comment
-
-
***> This is very likely to have parallels with control efforts for CWD in cervids.
Rapid recontamination of a farm building occurs after attempted prion removal
Kevin Christopher Gough, BSc (Hons), PhD1, Claire Alison Baker, BSc (Hons)2, Steve Hawkins, MIBiol3, Hugh Simmons, BVSc, MRCVS, MBA, MA3, Timm Konold, DrMedVet, PhD, MRCVS3 and Ben Charles Maddison, BSc (Hons), PhD2
Author affiliations
School of Veterinary Medicine and Science, The University of Nottingham, Loughborough, UK ADAS, School of Veterinary Medicine and Science, The University of Nottingham, Loughborough, UK Animal Sciences Unit, Pathology Department, Animal & Plant Health Agency Weybridge, New Haw, Addlestone, Surrey, UK E-mail for correspondence; ben.maddison@adas.co.uk
Abstract
The transmissible spongiform encephalopathy scrapie of sheep/goats and chronic wasting disease of cervids are associated with environmental reservoirs of infectivity.
Preventing environmental prions acting as a source of infectivity to healthy animals is of major concern to farms that have had outbreaks of scrapie and also to the health management of wild and farmed cervids.
Here, an efficient scrapie decontamination protocol was applied to a farm with high levels of environmental contamination with the scrapie agent.
Post-decontamination, no prion material was detected within samples taken from the farm buildings as determined using a sensitive in vitro replication assay (sPMCA).
A bioassay consisting of 25 newborn lambs of highly susceptible prion protein genotype VRQ/VRQ introduced into this decontaminated barn was carried out in addition to sampling and analysis of dust samples that were collected during the bioassay.
Twenty-four of the animals examined by immunohistochemical analysis of lymphatic tissues were scrapie-positive during the bioassay, samples of dust collected within the barn were positive by month 3.
The data illustrates the difficulty in decontaminating farm buildings from scrapie, and demonstrates the likely contribution of farm dust to the recontamination of these environments to levels that are capable of causing disease.
snip...
PrPC is ubiquitous in its distribution in vivo2 and with both scrapie and CWD the in vivo dissemination of infectivity is also widespread with PrPSc usually accumulating within peripheral lymphatic tissues before the CNS.3 4 With scrapie, PrPSc can be secreted/ excreted via a multiplicity of routes including saliva,5 6 milk,7 faeces,8 skin9 and urine.10 The accumulation of this material within the environment (particularly the built farm environment),11 12 creates levels of infectivity that can be transmitted to naïve animals. These reservoirs of infectivity can remain infectious for prolonged periods of time, in one such recorded incident at least 16 years.13 The advent of high sensitivity prion replication assays such as protein misfolding cyclic amplification (PMCA) with application to sheep/goat scrapie14 15 has allowed the monitoring of prions within environments.11
Attempts to decontaminate pens on a scrapie-affected farm and measuring efficacy using a sheep bioassay were previously reported.12 It was concluded that the failure of effective decontamination within that study was likely to have been due to the incomplete farm decontamination and the presence of dust containing infectious prions that recontaminated the pen surfaces. The serial protein misfolding cyclic amplification (sPMCA) technique was recently used to confirm the presence of prions within extracts prepared from dust samples that had settled on sterile surfaces.16 Given the presence of mobile infectious prions within dust, it was proposed that for effective scrapie decontamination emphasis should be given to the removal of all sources of dust within the decontamination strategy for a farm. More recently, the sPMCA technique has been used by the authors' laboratory to look at effective methods of decontaminating prions bound to concrete surfaces within the laboratory setting.17 This study demonstrated that current methodology based on a one-hour exposure to 20000 ppm free chlorine was likely to be ineffective at removing surface-bound scrapie prion. However, there was an enhanced effectiveness of this chemical decontamination when using multiple applications over four hours. Here, a study is described where a scrapie-affected farm was decontaminated using four applications of 20000 ppm free chlorine to livestock barns and concreted areas. The decontamination also included a high-level clean of the buildings that had housed sheep to remove all traces of dust as far as practicable before the chemical decontamination procedure. Following these treatments the surfaces within the barn were demonstrably free from prion using a sensitive sPMCA assay. The presence of any residual infectivity was then evaluated by sheep bioassay and dust samples collected during the bioassay were assayed for prion seeding activity by sPMCA.
snip...
Discussion
The authors' previous work on this farm indicated that dust harbours low levels of mobile scrapie prions that can accumulate on surfaces16 and this is likely to perpetuate transmission of scrapie within such a farm environment.12 In addition, previous in vitro modelling of scrapie prions bound to a concrete ‘fomite’ demonstrated that prion seeding activity could be inactivated by four applications of 20,000 ppm free chlorine as measured by a sPMCA assay. This previous modelling demonstrated that residual contamination of the swab extract with hypochlorite at levels which would inhibit the sPMCA are unlikely, and the authors consider these results as reduction in seeding titre.17 Here, this same decontamination methodology was tested within a farm-scale study which also included steps to remove dust within the barns. This study demonstrated that this thorough decontamination method applied to a farm with a high incidence of naturally acquired scrapie was sufficient to remove scrapie prions on surfaces to levels that were undetectable by sPMCA, one of the most sensitive biochemical assays for prions. The authors' sPMCA assay has an limit of detection of around 1–10pg scrapie-infected sheep brain per sPMCA reaction. The authors assume that the samples negative by sPMCA had less than this amount (of brain equivalent) within the extracts that were prepared. This treatment together with measures designed to minimise the amount of dust retained within the buildings (vacuuming all surfaces, pressure washing and then hypochlorite treatment) was expected to have removed all infectivity from the buildings and the concrete areas surrounding them, and it was anticipated that the sheep bioassay would confirm absence of infective prion.
However, the introduction into this decontaminated barn of 25 VRQ/VRQ sheep (a genotype highly susceptible to classical scrapie) demonstrated that all animals, with the exception of 1 lamb that died at 122 dpe, had detectable PrPSc in lymphoid tissue, indicating infection with the scrapie agent. This included 14 animals (54 per cent) that were PrPSc-positive on the first RAMALT analysis at 372 dpe or 419 dpe. Although infected sheep were removed based on a positive RAMALT result, it is possible that lateral transmission or subsequent contamination of the environment from infected sheep had contributed to the rapid spread of scrapie in nearly all sheep. It has been shown previously that objects in contact with scrapie-infected sheep, such as water troughs and fence posts, can act as a reservoir for infection.23 As in the authors' previous study,12 the decontamination of this sheep barn was not effective at removing scrapie infectivity, and despite the extra measures brought into this study (more effective chemical treatment and removal of sources of dust) the overall rates of disease transmission mirror previous results on this farm. With such apparently effective decontamination (assuming that at least some sPMCA seeding ability is coincident with infectivity), how was infectivity able to persist within the environment and where does infectivity reside? Dust samples were collected in both the bioassay barn and also a barn subject to the same decontamination regime within the same farm (but remaining unoccupied). Within both of these barns dust had accumulated for three months that was able to seed sPMCA, indicating the accumulation of scrapie-containing material that was independent of the presence of sheep that may have been incubating and possibly shedding low amounts of infectivity.
This study clearly demonstrates the difficulty in removing scrapie infectivity from the farm environment. Practical and effective prion decontamination methods are still urgently required for decontamination of scrapie infectivity from farms that have had cases of scrapie and this is particularly relevant for scrapiepositive goatherds, which currently have limited genetic resistance to scrapie within commercial breeds.24 This is very likely to have parallels with control efforts for CWD in cervids.
Acknowledgements The authors thank the APHA farm staff, Tony Duarte, Olly Roberts and Margaret Newlands for preparation of the sheep pens and animal husbandry during the study. The authors also thank the APHA pathology team for RAMALT and postmortem examination.
Funding This study was funded by DEFRA within project SE1865.
Competing interests None declared.
Saturday, January 5, 2019
Rapid recontamination of a farm building occurs after attempted prion removal
FRIDAY, JANUARY 11, 2019
TEXAS TAHC Legislative Appropriations Request For Fiscal Years 2018 and 2019 CWD TSE PRION PAY TO PLAY PROGRAM
Comment
-
Look at the bright side!....The price of leases is going to drop when nobody will want to eat those CWD contaminated deer.
You have plenty of copy and paste articles by flounder, but ask him to post the fatalities that are mounting with humans dying from it! Chances are he can't!
It's nothing new, or something that just popped up out of nowhere!
I can't prove that it's not a concern to a degree, but I wouldn't put it past the PETA types to take this approach as a way to stop hunting via a fear agenda, and one that even the TPWD upper management has bought in to.Last edited by Jimbo47; 01-12-2019, 12:50 PM.Comment
-
Researchers seem to be making some progress.
Comment
-
Originally posted by lab man View Post
Good read labman!
Never knew that CWD was in Norway and South Korea! Must be those deer breeders from Texas fault.
Sent from my iPhone using TapatalkComment
-
What about the transfer to pigs, cows, etc......Comment
-
-
Cwd and scrapie transmits to pigs by oral route
cwd scrapie pigs oral routesOriginally posted by Ruggedhunts45 View Post
***> However, at 51 months of incubation or greater, 5 animals were positive by one or more diagnostic methods. Furthermore, positive bioassay results were obtained from all inoculated groups (oral and intracranial; market weight and end of study) suggesting that swine are potential hosts for the agent of scrapie. <***
>*** Although the current U.S. feed ban is based on keeping tissues from TSE infected cattle from contaminating animal feed, swine rations in the U.S. could contain animal derived components including materials from scrapie infected sheep and goats. These results indicating the susceptibility of pigs to sheep scrapie, coupled with the limitations of the current feed ban, indicates that a revision of the feed ban may be necessary to protect swine production and potentially human health. <***
***> Results: PrPSc was not detected by EIA and IHC in any RPLNs. All tonsils and MLNs were negative by IHC, though the MLN from one pig in the oral <6 month group was positive by EIA. PrPSc was detected by QuIC in at least one of the lymphoid tissues examined in 5/6 pigs in the intracranial <6 months group, 6/7 intracranial >6 months group, 5/6 pigs in the oral <6 months group, and 4/6 oral >6 months group. Overall, the MLN was positive in 14/19 (74%) of samples examined, the RPLN in 8/18 (44%), and the tonsil in 10/25 (40%).
***> Conclusions: This study demonstrates that PrPSc accumulates in lymphoid tissues from pigs challenged intracranially or orally with the CWD agent, and can be detected as early as 4 months after challenge. CWD-infected pigs rarely develop clinical disease and if they do, they do so after a long incubation period.
This raises the possibility that CWD-infected pigs could shed prions into their environment long before they develop clinical disease.
Furthermore, lymphoid tissues from CWD-infected pigs could present a potential source of CWD infectivity in the animal and human food chains.
***> However, at 51 months of incubation or greater, 5 animals were positive by one or more diagnostic methods. Furthermore, positive bioassay results were obtained from all inoculated groups (oral and intracranial; market weight and end of study) suggesting that swine are potential hosts for the agent of scrapie. <***
>*** Although the current U.S. feed ban is based on keeping tissues from TSE infected cattle from contaminating animal feed, swine rations in the U.S. could contain animal derived components including materials from scrapie infected sheep and goats. These results indicating the susceptibility of pigs to sheep scrapie, coupled with the limitations of the current feed ban, indicates that a revision of the feed ban may be necessary to protect swine production and potentially human health. <***
Scrapie Transmits To Pigs By Oral Route, what about the terribly flawed USA tse prion feed ban?
Research Project: Pathobiology, Genetics, and Detection of Transmissible Spongiform Encephalopathies Location: Virus and Prion Research
2017 Annual Report
1a. Objectives (from AD-416):
snip...
However, at 51 months of incubation or greater, 5 animals were positive by one or more diagnostic methods. Furthermore, positive bioassay results were obtained from all inoculated groups (oral and intracranial; market weight and end of study) suggesting that swine are potential hosts for the agent of scrapie. Although the current U.S. feed ban is based on keeping tissues from TSE infected cattle from contaminating animal feed, swine rations in the U.S. could contain animal derived components including materials from scrapie infected sheep and goats. These results indicating the susceptibility of pigs to sheep scrapie, coupled with the limitations of the current feed ban, indicates that a revision of the feed ban may be necessary to protect swine production and potentially human health.
2. Determined that pigs naturally exposed to chronic wasting disease (CWD) may act as a reservoir of CWD infectivity. Chronic wasting disease is a naturally occurring, fatal, neurodegenerative disease of cervids. The potential for swine to serve as a host for the agent of CWD disease is unknown. The purpose of this study was to investigate the susceptibility of swine to the CWD agent following experimental oral or intracranial inoculation. Pigs were assigned to 1 of 3 groups: intracranially inoculated; orally inoculated; or non-inoculated. At market weight age, half of the pigs in each group were tested ('market weight' groups). The remaining pigs ('aged' groups) were allowed to incubate for up to 73 months post inoculation (MPI). Tissues collected at necropsy were examined for disease-associated prion protein (PrPSc) by multiple diagnostic methods. Brain samples from selected pigs were bioassayed in mice expressing porcine prion protein. Some pigs from each inoculated group were positive by one or more tests. Bioassay was positive in 4 out of 5 pigs assayed. Although only small amounts of PrPSc were detected using sensitive methods, this study demonstrates that pigs can serve as hosts for CWD. Detection of infectivity in orally inoculated pigs using mouse bioassay raises the possibility that naturally exposed pigs could act as a reservoir of CWD infectivity. Currently, swine rations in the U.S. could contain animal derived components including materials from deer or elk. In addition, feral swine could be exposed to infected carcasses in areas where CWD is present in wildlife populations. The current feed ban in the U.S. is based exclusively on keeping tissues from TSE infected cattle from entering animal feeds. These results indicating the susceptibility of pigs to CWD, coupled with the limitations of the current feed ban, indicates that a revision of the feed ban may be necessary to protect swine production and potentially human health.
3. Developed a method for amplification and discrimination of the 3 forms of BSE in cattle. The prion protein (PrP) is a protein that is the causative agent of transmissible spongiform encephalopathies (TSEs). The disease process involves conversion of the normal cellular PrP to a pathogenic misfolded conformation. This conversion process can be recreated in the lab using a misfolding amplification process known as real-time quaking induced conversion (RT-QuIC). RT-QuIC allows the detection of minute amounts of the abnormal infectious form of the prion protein by inducing misfolding in a supplied substrate. Although RT-QuIC has been successfully used to detect pathogenic PrP with substrates from a variety of host species, prior to this work bovine prion protein had not been proven for its practical uses for RT-QuIC. We demonstrated that prions from transmissible mink encephalopathy (TME) and BSE-infected cattle can be detected with using bovine prion proteins with RT-QuIC, and developed an RT-QuIC based approach to discriminate different forms of BSE. This rapid and robust method, both to detect and discriminate BSE types, is of importance as the economic implications for different types of BSE vary greatly.
FRIDAY, APRIL 20, 2018
*** Scrapie Transmits To Pigs By Oral Route, what about the terribly flawed USA tse prion feed ban?
Research Project: Pathobiology, Genetics, and Detection of Transmissible Spongiform Encephalopathies
Comment
-
Originally posted by kyle1974 View Post
or, feed, grains, hay, straw, etc...
it's getting ready to get real nasty folks, and yes, Norway, South Korea, and FINLAND, have documented CWD, with South Korea CWD coming from Canada, via USA, and i would bet Norway and Finland got it the same way. they imported if from North America some how, imo.
Subject: Norway cwd hay and straw
Regulatory Process
New rules on additional requirements for imports of hay and straw for animal feed
Published 19.09.2018 Last modified 08.11.2018 Print
The crop failure after the drought this summer means that many farmers and others who keep livestock need to supplement with purchased hay and straw. Purchasing roughage from other countries will always entail a risk of introducing unwanted infectious agents and plants, which can lead to a health hazard for humans, animals and plants.
A regulation is proposed with additional requirements for imports of hay and straw into animal feed from countries outside the EEA. High and straw imported into Norway as animal feed must:
is accompanied by a certificate from a public veterinarian in the country of origin stating that the product has been harvested from an area where there are no restrictions due to contagious animal disease,
is accompanied by a confirmation from the manufacturer that the product has been harvested from farms where it is not fertilized with manure during the last two years,
is accompanied by a confirmation from the manufacturer that the product has been stored for at least two months in the consignment country upon import or is stored in Norway for two months before use.
High and straw from the US and Canada must also be accompanied by a certificate from a public veterinarian stating that the product has been harvested in states or provinces where Chronic Wasting Disease has not been detected on cervids.
The Norwegian Food Safety Authority holds an open consultation meeting on the proposal on Wednesday, 26 September, at 10.30. The meeting is held in the auditorium of the Norwegian Food Safety Authority's head office premises in Oslo (Ullevålsveien 76).
The time in the work is described in the timeline below and the black box shows where we are in the process
Timeline for this work:
Consultation
The Norwegian Food Safety Authority sends proposals for new rules on additional requirements for imports of hay and straw for animal feed on consultation.
***> NORWAY CWD UPDATE December 2018
Report from the Norwegian Scientific Committee for Food and Environment (VKM) 2018: 16
Factors that can contribute to spread of CWD – an update on the situation in Nordfjella, Norway
Opinion of Panel on biological hazards of the Norwegian Scientific Committee for Food and Environment
13.12.2018
ISBN: 978-82-8259-316-8
ISSN: 2535-4019
Norwegian Scientific Committee for Food and Environment (VKM)
Po 222 Skøyen
0213 Oslo
Norway
FRIDAY, DECEMBER 14, 2018
Norway, Nordfjella VKM 2018 16 Factors that can contribute to spread of CWD TSE Prion UPDATE December 14, 2018
THURSDAY, OCTOBER 25, 2018
***> Norway New additional requirements for imports of hay and straw for animal feed from countries outside the EEA due to CWD TSE Prion
TUESDAY, JULY 03, 2018
Chronic Wasting Disease CWD TSE Prion Global Report Update, USA, CANADA, KOREA, NORWAY, FINLAND, Game Farms and Fake news
Subject: TEXAS BREEDER DEER ESCAPEE WITH CWD IN THE WILD, or so the genetics would show?
TEXAS BREEDER DEER ESCAPEE WITH CWD IN THE WILD, or so the genetics would show?
OH NO, please tell me i heard this wrong, a potential Texas captive escapee with cwd in the wild, in an area with positive captive cwd herd?
apparently, no ID though. tell me it ain't so please...
23:00 minute mark
''Free Ranging Deer, Dr. Deyoung looked at Genetics of this free ranging deer and what he found was, that the genetics on this deer were more similar to captive deer, than the free ranging population, but he did not see a significant connection to any one captive facility that he analyzed, so we believe, Ahhhhhh, this animal had some captive ahhh, whatnot.''
Texas Chronic Wasting Disease CWD TSE Prion Symposium 2018 posted January 2019 VIDEO SET 18 CLIPS
See Wisconsin update...terrible news, right after Texas updated map around 5 minute mark...
update on Wisconsin from Tammy Ryan...
[ame="https://www.youtube.com/watch?v=hvy2SMGQt6o&index=11&list=PL7ZG8MkruQh3wI9 6XQ8_EymytO828rGxj"]Texas CWD Symposium: CWD Management & Response in Wisconsin - YouTube[/ame]
Wyoming CWD Dr. Mary Wood
''first step is admitting you have a problem''
''Wyoming was behind the curve''
wyoming has a problem...
[ame="https://www.youtube.com/watch?v=y1bsK4Igt1o&index=10&list=PL7ZG8MkruQh3wI9 6XQ8_EymytO828rGxj"]Texas CWD Symposium: CWD in Wyoming 30+ Yrs Later - YouTube[/ame]
SATURDAY, JANUARY 19, 2019
Texas Chronic Wasting Disease CWD TSE Prion Symposium 2018 posted January 2019 VIDEO SET 18 CLIPS
TUESDAY, JANUARY 29, 2019
TEXAS REPORTS 2 MORE CWD TSE PRION ALL WILD CERVID TOTAL TO DATE 141
http:// https://tpwd.texas.gov/huntwild/wild.../cwd/tracking/
terryComment
-
Originally posted by Texans42 View Post
i tried to tell them they have been waltzing across the border from wsmr trans pecos region year after year since about 2001 or 2002 up until 2012, where they detected cwd in the wild...right where i had been trying to warn them.
CWD SAMPLING TEXAS (but NOT in the obvious place, the NM, TEXAS border) December 12, 2003 at 2:15 pm:
you can't say i did not try and warn them...17 years ago, 2002. but now you have Arkansas, and Louisiana hasn't a clue on cwd or Oklahoma, with their sample survey figures to date imo...terry
*** Subject: CWD testing in Texas ***
Date: Sun, 25 Aug 2002 19:45:14 –0500
From: Kenneth Waldrup
To: flounder@wt.net
snip...see ;
i am not a Doctor and never anywhere have i implied i was, or have implied nothing more than who i am, and why i do it...terry
Subject: CWD SURVEILLANCE STATISTICS TEXAS (total testing figures less than 50 in two years) Date: Sun, 25 Aug 2002 21:06:49 –0700
From: "Terry S. Singeltary Sr."
Reply-To: Bovine Spongiform Encephalopathy
To: BSE-L@uni-karlsruhe.de
######## Bovine Spongiform Encephalopathy #########
greetings list members,
here are some figures on CWD testing in TEXAS...TSS
Dear Dr. Singletary,
In Fiscal Year 2001, seven deer from Texas were tested by the National Veterinary Services Laboratory (NVSL) for CWD (5 fallow deer and 2 white-tailed deer). In Fiscal Year 2002, seven elk from Texas were tested at NVSL (no deer). During these two years, an additional six elk and one white-tailed deer were tested at the Texas Veterinary Medical Diagnostic Laboratory (TVMDL). In Fiscal Year 2002, four white-tailed deer (free-ranging clinical suspects) and at least eight other white-tailed deer have been tested at TVMDL. One elk has been tested at NVSL. All of these animals have been found negative for CWD. Dr. Jerry Cooke of the Texas Parks and Wildlife Department also has records of 601 clinically ill white-tailed deer which were necropsied at Texas A&M during the late 1960's and early 1970's, and no spongiform encepalopathies were noted. Thank you for your consideration.
******x
Texas Animal Health Commission
(personal communication...TSS)
Austin 8 news
snip...
"There's about 4 million deer in the state of Texas, and as a resource I think we need to be doing as much as we can to look for these diseases," said Doug Humphreys with Texas Parks and Wildlife. "Right now Texas is clear. We haven't found any, but that doesn't mean we don't look."
With approximately 4 million animals, Texas has the largest population of white-tailed deer in the nation. In addition, about 19,000 white-tailed deer and 17,000 elk are being held in private facilities. To know if CWD is present in captive herds, TPWD and Texas Animal Health Commission are working with breeders to monitor their herds.
How is it spread?
It is not known exactly how CWD is spread. It is believed that the agent responsible for the disease may be spread both directly (animal to animal contact) and indirectly (soil or other surface to animal). It is thought that the most common mode of transmission from an infected animal is via saliva, feces, and urine.
some surveillance?
beyond the _potential_ methods of transmissions above, why, not a single word of SRM of various TSE species in feed as a source?
it's a known fact they have been feeding the deer/elk the same stuff as cows here in USA.
and the oral route has been documented of CWD to mule deer fawns in lab studies.
not to say that other _potential_ transmission mechanisms are possible, but why over look the obvious?
TSS
########### http://mailhost.rz.uni-karlsruhe.de/warc/bse-l.html ############
From: Ken Waldrup, DVM, PhD (host25-207.tahc.state.tx.us)
Subject: Re: CWD SAMPLING TEXAS (but NOT in the obvious place, the NM, TEXAS border)
Date: December 15, 2003 at 3:43 pm PST
In Reply to: CWD SAMPLING TEXAS (but NOT in the obvious place, the NM, TEXAS border) posted by TSS on December 12, 2003 at 2:15 pm:
Dear sirs:
With regard to your comment about Texas NOT looking for CWD along the New Mexico border, it is painfully obvious that you do not know or understand the natural distribution of mule deer out there or the rights of the land owners in this state. As of 15 December 2003, a total of 42 deer had been sampled from what we call "Trans-Pecos", beyond the Pecos River. Mule deer are very widely dispersed through this area, sometimes at densities of one animal per 6 square miles. The Texas Parks and Wildlife Department does not have the legal authority to trepass on private property to collect deer. Some landowners are cooperative. Some are not. Franklin State Park is at the very tip of Texas, and deer from the park have been tested (all negative). One of the single largest land owners along the border is the National Park Service. Deer and elk from the Guadalupe Peak National Park cannot be collected with federal permission. The sampling throughout the state is based on the deer populations by eco-region and is dictated by the availability of funds. I am concerned about your insinuation that CWD is a human health risk. We are at a stand-off - you have no proof that it is and I have no definitive proof that it isn't. However I would say that the inferred evidence from Colorado, Wyoming and Wisconsin suggests that CWD is not a human health concern (i.e. no evidence of an increased incidence of human brain disorders within the CWD "endemic" areas of these states). From my professional interactions with the Texas Parks and Wildlife Department, I can definitely say that they want to do a thorough and sound survey throughout the state, not willy-nilly "look here, look there". There are limitations of manpower, finances and, in some places, deer populations. I would congratulate TPWD for doing the best job with the limitations at hand rather than trying to browbeat them when you obviously do not understand the ecology of West Texas. Thank you for your consideration.
======================
From: TSS (216-119-139-126.ipset19.wt.net)
Subject: Re: CWD SAMPLING TEXAS (but NOT in the obvious place, the NM, TEXAS border)
Date: December 16, 2003 at 11:03 am PST
In Reply to: Re: CWD SAMPLING TEXAS (but NOT in the obvious place, the NM, TEXAS border) posted by Ken Waldrup, DVM, PhD on December 15, 2003 at 3:43 pm:
HEllo Dr. Waldrup,
thank you for your comments and time to come to this board.
Ken Waldrup, DVM, PhD states;
> it is painfully obvious that you do not know or understand the natural distribution of mule deer out there or the rights of the land owners in this state...
TSS states;
I am concerned about all deer/elk not just mule deer, and the rights of land owners (in the case with human/animal TSEs) well i am not sure of the correct terminology, but when the States deer/elk/cattle/sheep/humans are at risk, there should be no rights for land owners in this case. the state should have the right to test those animals. there are too many folks out there that are just plain ignorant about this agent. with an agent such as this, you cannot let landowners (and i am one) dictate human/animal health, especially when you cannot regulate the movement of such animals...
snip...see;
terryComment
Comment