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CWD found in panhandle whitetail

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    Originally posted by critter69 View Post
    Could be a number of ways, one way, someone from there could have hunted in let's say Colorado ( somewhere that it exist. ) transported the animal back to Noway. Processed it, threw the waste out in the back forty. That's really all it would take. Maybe other scenarios that are even easier, birds migrating who knows. It's in other places over seas also, that have imported deer and elk from the us.
    And this is exactly how this is about to get really sticky... Disposal by incineration is already being talked about. It was talked about several years ago as a matter of fact, by TPWD.
    Processors and taxidermists will be on the verge of being regulated out of business when this thing busts wide open and the panic and overreaction occurs.. It won't be long now..

    No more processing your own deer and discarding the waste on your property.. Nope. SMH.
    Last edited by PondPopper; 01-11-2018, 09:17 PM.

    Comment


      Originally posted by DTala View Post
      WT Deer were imported into Finland, but that was in 1930's before Ft Collins.

      A CWD positive deer from White Sands NM was touted as the proof that CWD was "naturally occurring" for years and years....till it was discovered that someone actually turned mule deer loose there from Ft Collins area.
      Where is the documentation on white sands of a release post 1967?

      Comment


        Originally posted by sideways View Post
        If I eat a deer with CWD will I die?


        Sent from my iPhone using Tapatalk Pro
        If I don’t eat a deer with CWD, will I not die?
        Have we philosophically discovered the key to immortality?

        I don’t have links or research to back this, but I’m pretty sure there are documented cases of other prion diseases jumping species (Mad Cow was a prion disease, didn’t it take some folks down?). I wonder about the incubation period, relative to the average lifespan of the critter carrying it. I know the incubation period for deer is one to two years before any symptoms are seen, but think about how long (or short) deer live anyway, a buck reaching age ten might as well be a unicorn. Would it be feasible to see the incubation period in people be much longer? If I may really abuse the notion of averages for a moment, and we set the average lifespan of a whitetail at ten years, and apply an incubation period of 2 years, that’s 20% of the deer’s potential lifespan. If we scale that to humans, who commonly see 100 year lifespans, could the idea of a twenty-year incubation period be feasible?

        Comment


          Also remember that these prion diseases (Mad Cow, Kuru, and Scrapie) mainly showed up in human populations that ate central nervous system tissue or meat that had been contaminated with it.

          That's why we changed the beef industry after the mad cow scare in Britain.

          Comment


            From the "chronic wasting disease alliance"
            The origin of CWD is unknown, and it may never be possible to definitively determine how or when CWD arose. It was first recognized as a syndrome in captive mule deer held in wildlife research facilities in Colorado in the late 1960s, but it was not identified as a TSE until the 1970s. Computer modeling suggests the disease may have been present in free-ranging populations of mule deer for more than 40 years.

            Scrapie, a TSE of domestic sheep, has been recognized in the United States since 1947, and it is possible that CWD was derived from scrapie. It is possible, though never proven, that deer came into contact with scrapie-infected sheep either on shared pastures or in captivity somewhere along the front range of the Rocky Mountains, where high levels of sheep grazing occurred in the early 1900s.

            It may be possible that CWD is a spontaneous TSE that arose in deer in the wild or in captivity and has biological features promoting transmission to other deer and elk


            and this from a popular press article
            “I think Gene’s hypothesis is very reasonable. I, too, would lean toward scrapie, but there’s nothing to prove it is how this disease first began,” said Beth Williams, now a professor at the veterinary science lab at the University of Wyoming in Laramie.

            Williams said she asked around about sheep with scrapie being on the campus in the late 1960s and couldn’t find anyone who was aware of any such project.

            “However, they didn’t always diagnose scrapie so I wouldn’t rule it out,” she added.

            Wildlife division veterinarian Mike Miller, who has done extensive study on CWD, doubts Schoonveld’s theory.

            “In all the literature we have searched there never has been a mention of scrapie in sheep in those pens during that period. And even if there was, there is nothing to prove CWD is the result of a transfer of scrapie from infected sheep to deer or elk,” Miller said.

            Comment


              Originally posted by Aggie PhD View Post
              From the "chronic wasting disease alliance"
              The origin of CWD is unknown, and it may never be possible to definitively determine how or when CWD arose. It was first recognized as a syndrome in captive mule deer held in wildlife research facilities in Colorado in the late 1960s, but it was not identified as a TSE until the 1970s. Computer modeling suggests the disease may have been present in free-ranging populations of mule deer for more than 40 years.

              Scrapie, a TSE of domestic sheep, has been recognized in the United States since 1947, and it is possible that CWD was derived from scrapie. It is possible, though never proven, that deer came into contact with scrapie-infected sheep either on shared pastures or in captivity somewhere along the front range of the Rocky Mountains, where high levels of sheep grazing occurred in the early 1900s.

              It may be possible that CWD is a spontaneous TSE that arose in deer in the wild or in captivity and has biological features promoting transmission to other deer and elk


              and this from a popular press article
              “I think Gene’s hypothesis is very reasonable. I, too, would lean toward scrapie, but there’s nothing to prove it is how this disease first began,” said Beth Williams, now a professor at the veterinary science lab at the University of Wyoming in Laramie.

              Williams said she asked around about sheep with scrapie being on the campus in the late 1960s and couldn’t find anyone who was aware of any such project.

              “However, they didn’t always diagnose scrapie so I wouldn’t rule it out,” she added.

              Wildlife division veterinarian Mike Miller, who has done extensive study on CWD, doubts Schoonveld’s theory.

              “In all the literature we have searched there never has been a mention of scrapie in sheep in those pens during that period. And even if there was, there is nothing to prove CWD is the result of a transfer of scrapie from infected sheep to deer or elk,” Miller said.
              Thats very interesting. Seems the amount of misinformation about CWD is vast. Unfortunate as it complicates intelligent response and invokes unnecessary emotional reactions

              Comment


                This popped up on my facebook timeline yesterday. Thought some would find it interesting. Sorry if it has already been posted.

                A study in which macaques contracted chronic wasting disease from eating CWD-positive deer meat has heightened concerns about human susceptibility to the disease.


                Macaque monkeys contracted chronic wasting disease after eating meat from CWD-positive deer, according to Canadian researchers.

                The findings are the first known transmissions of the prion disease to a primate from eating diseased venison and have heightened concerns of human susceptibility to CWD.

                “The assumption was for the longest time that chronic wasting disease was not a threat to human health,” said Stefanie Czub, prion researcher with the Canadian Food Inspection Agency, in remarks published Saturday in The Tyee, a Vancouver, British Columbia, magazine. "But with the new data, it seems we need to revisit this view to some degree."

                Czub is leading the project, which began in 2009 and is funded by Alberta Prion Research Institute at the University of Calgary.

                ADVERTISING

                Eighteen macaques have been exposed to CWD in various ways to study the transmission potential of the disease.

                Three of five macaques that were fed infected white-tailed deer meat over a three-year period tested positive for CWD.


                The meat fed to the macaques represented the human equivalent of eating a 7-ounce steak per month.

                Macaques that had the CWD prion injected into their brains also contracted the disease.

                Those that had infected material rubbed on their skin — designed to simulate contact a hunter might have while field dressing a deer — have not contracted the disease.

                Czub presented the results May 25 in a talk titled "CWD Transmission into non-human Primates" at the Prion 2017 conference in Edinburgh, Scotland.

                The finding of oral transmission of CWD to a primate through eating of infected meat is most troubling to scientists and conservationists.

                “This study does not mean people will get CWD. But it means people need to be considering that possibility.”

                Dave Clausen
                "This study does not mean people will get CWD," said Dave Clausen of Amery, a veterinarian, deer hunter and former chairman of the Wisconsin Natural Resources Board. "But it means people need to be considering that possibility."

                Wisconsin hunters and their family members likely have more contact with CWD each year than those in any other state or province.

                The contagious disease, caused by a misfolded prion, or protein, is found at prevalence rates exceeding 30% in parts of deer-rich southern Wisconsin.

                Last year, 442 of 3,758 deer submitted for testing in the southern farmland zone were CWD-positive. But more than 10 times as many deer were killed in the zone and not tested, many of which likely carried the disease.


                According to the Alliance for Public Wildlife, a Canadian-based wildlife conservation organization, hunting families in North America consume between 7,000 and 15,000 CWD-infected animals every year.

                Chronic wasting disease is fatal to deer, elk and moose. Since it is a transmissible spongiform encephalopathy (TSE) and in the same family as two diseases known to be fatal to humans (mad cow disease and Creutzfeldt-Jakob) researchers and health officials have long wanted to know more about the potential for interspecies CWD spread.

                Based on the macaque study, Health Canada issued an updated CWD risk advisory.

                "While extensive disease surveillance in Canada and elsewhere has not provided any direct evidence that CWD has infected humans, the potential for CWD to be transmitted to humans cannot be excluded," the advisory states. "In exercising precaution, (Health Canada) continues to advocate that the most prudent approach is to consider that CWD has the potential to infect humans."

                From the World Health Organization to the federal Centers for Disease Control to state agencies, health officials are united in their recommendations to avoid eating meat from a CWD-positive animal.

                Since 2003, the Wisconsin Department of Health Services (DHS) has issued a warning against consuming CWD-positive venison. The Canadian research reinforces the agency's position, said Jennifer Miller, DHS communications specialist.

                "The Wisconsin Department of Health Services is aware of the study involving macaques contracting chronic wasting disease after being fed meat from deer infected with CWD," said Miller. "The department will continue to encourage hunters to have deer tested that were harvested from areas of the state where CWD is known to exist."

                Miller said the department will also continue to discourage the consumption of meat from deer harvested anywhere that showed signs of illness — for example, deer that appeared emaciated or that acted abnormally.

                The agency's recommendation states "venison from deer harvested inside the CWD Management Zone should not be consumed or distributed to others until CWD test results on the source deer are known to be negative."

                The Wisconsin guideline says venison from multiple deer should be kept separate and labeled before freezing so any meat from a CWD-positive animal can be discarded.

                "Some might call the recommendation overly cautious," James Kazmierczak, DHS state public health veterinarian, told the Milwaukee Journal Sentinel in a July 2015 interview. "But the CWD prion has been found in muscle tissue. So from a public health perspective, the safest path is to test your deer and not consume meat from a CWD-positive animal."

                In its Deer Hunting 2016 Pamphlet, the Wisconsin Department of Natural Resources also advises hunters to not eat the eyes, brain, spinal cord, spleen, tonsils or lymph nodes of any deer; to wear rubber or latex gloves when field-dressing carcasses, and to bone out the meat from the animal.

                The Canadian study will likely wrap up in 2018, Czub said.

                The preliminary results have already caused many to take notice.

                "Research on prion diseases is unveiling new findings all the time," Clausen said. "This latest news confirms its wiser than ever to get your deer tested and follow the precautions."

                Comment


                  yikes!

                  Comment


                    Originally posted by Aggie PhD View Post


                    and this from a popular press article
                    “I think Gene’s hypothesis is very reasonable. I, too, would lean toward scrapie, but there’s nothing to prove it is how this disease first began,” said Beth Williams, now a professor at the veterinary science lab at the University of Wyoming in Laramie.

                    Williams said she asked around about sheep with scrapie being on the campus in the late 1960s and couldn’t find anyone who was aware of any such project.

                    “However, they didn’t always diagnose scrapie so I wouldn’t rule it out,” she added.

                    Wildlife division veterinarian Mike Miller, who has done extensive study on CWD, doubts Schoonveld’s theory.

                    “In all the literature we have searched there never has been a mention of scrapie in sheep in those pens during that period. And even if there was, there is nothing to prove CWD is the result of a transfer of scrapie from infected sheep to deer or elk,” Miller said.

                    those two statements are a premeditated ****n lie.

                    Comment


                      not so much misinformation as a coverup of the FUBAR at FT Collins in the 60's

                      Comment


                        https://youtu.be/Vtt1kAVDhDQ Here's some more, I posted this some time back after watching it. Along with other articles, one of which some body already posted. This video is like 7 or 8 hours if I remember. I watched it over a several day period.

                        Comment


                          Originally posted by TildenHunter View Post
                          It has been around long before high fences.....this statement makes you sound uniformed
                          That statement I am not buying at all. High fence zoos have been around since at least the early 80s that I know of. Never heard of CWD, until about 8 to 10 years ago.

                          Comment


                            Originally posted by RifleBowPistol View Post
                            That statement I am not buying at all. High fence zoos have been around since at least the early 80s that I know of. Never heard of CWD, until about 8 to 10 years ago.


                            First identified in the 60s.....


                            Sent from my iPhone using Tapatalk

                            Comment


                              Prolly caused by anther restriction. Shoot them all young before they get old enough to get sick. Reminds me of the bangs test for cattle back 15-20 years ago. There is not much telling how many cows were tested positive as having bangs that really did have bangs and were branded with a big b and slaughtered. Lots of ranches had their ranches whole herd slaughtered According to a biologist I talked to the cwd test isn't that reliable anyways. Good thing the deer belong to the state of Texas.

                              Comment


                                Originally posted by RifleBowPistol View Post
                                That statement I am not buying at all. High fence zoos have been around since at least the early 80s that I know of. Never heard of CWD, until about 8 to 10 years ago.
                                Actually it was found in a mule deer @ a Canadian Zoo in 1981.

                                Here's good timeline from Chronic Wasting Disease Alliance http://cwd-info.org/timeline/

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